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Repolarization after a test depolarization consistently evoked a transient inward “tail current” (ITail) that flowed via a second population of ion channels with poor (3 fold) K+ versus Na+ selectivity. The activity of these channels was increased by quinidine, 4 AP and progesterone. Vm in human sperm is therefore dependent upon a hyperpolarizing K+ current that flows via channels that most closely resemble those encoded by Slo3.
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